Alcoholic Cardiomyopathy: Causes, Symptoms, and Diagnosis

Guppy LJ, Littleton JM. Effect of calcium, Bay K 8644, and reduced perfusion on basic indices of myocardial function in isolated hearts from rats after prolonged exposure to ethanol. Kupari M. Acute cardiovascular effects of ethanol A controlled non-invasive study. Basic research studies have described an abundance of mechanisms that could underscore the functional and structural alterations found in ACM. Because of this, their origin could be multifactorial and linked both to the alcohol molecule and to its main metabolite, acetaldehyde.

In the absence of myocardial biopsy, the diagnosis of myocarditis is always questionable. However, given the characteristic rise and fall of cardiac enzyme levels, this supports the diagnosis of acute alcohol-induced myocardial damage. After one month of abstaining from alcohol, the patient was asymptomatic. A repeat echocardiogram revealed normal left ventricular function, with an ejection fraction of 62% by modified Simpson’s biplane method. The end-systolic dimension was 3.3 cm and the end-diastolic dimension was 4.8 cm . Alcohol can be safely removed from the body through medical detox where an individual can be monitored around the clock for several days to ensure safety and compliance.

Study design:

There are several things a doctor can do to treat alcoholic cardiomyopathy. The thing to remember is that if you keep on drinking heavily, your doctor’s treatment probably won’t help you much. Kupari M, Koskinen P. Relation of left ventricular function to habitual alcohol consumption. Guillo et al in 1997 described alcoholic cardiomyopathy recovery time the evolution of 9 ACM patients who had been admitted. He divided this cohort into two groups according to the evolution of the ejection fraction during 36 mo in which no deaths were recorded. The 6 subjects who experienced a clear improvement in their ejection fraction had fully refrained from drinking.

alcoholic cardiomyopathy recovery time

All-cause mortality was assessed using Kaplan–Meier survival curves, and the risk factors were assessed using Cox regression. A receiver operating characteristic curve analysis was performed to optimize the cutoff point for discriminating between the 2 risk groups. The doctor will also likely ask about your medical history, including your history of alcohol use. While lab testing won’t lead to an alcoholic cardiomyopathy diagnosis specifically, it can aid in identifying organ damage. Oftentimes X-rays and CT scans can detect an enlarged heart and congestion or fluid in the lungs while electrocardiograms may identify disruption in the heartbeat. Individuals at this point are in a serious position where the stakes are high, and they need the best clinical and therapeutic care they can get. Alcoholic Cardiomyopathy is the most serious concerns of the effects of alcohol on the heart.

How Does Alcoholic Cardiomyopathy Occur?

Constant medical supervision, medication, and psychological support can make the process safer and more comfortable. Ventricular assist devices are used to improve blood flow through the heart. VADs can be used for the long term or as a short-term intervention while the patient awaits a heart transplant. If a person is in end-stage heart failure and other treatments aren’t work, doctors may recommend a heart transplant.